D-AGING

D-Aging with Telomerase Activation (TA)

D-Aging: The Big Picture

Aging is a harsh reality. Our bodily systems decline at the rate of 1% per year (or more!) from their youthful peak, but through a scientific breakthrough that has evolved from the Traditional Chinese herb Astragalus, this steady decline can be stemmed.

Initiating the D-Aging Process with TA

In our quest to turn back the hands of time and give us the means to slow our aging process, T.A. Sciences is introducing a new D-Aging protocol: the equivalent of the fountain of youth on a cellular level. Telomerase Activation is a science-based concept about slowing the signs of aging, i.e. D-Aging.

Postponing the vital signs of aging is not an impossible dream. There really is an escape from the revolving door of anti-aging products and schemes. The new and exciting Patton Protocol is based on breakthrough science. Nothing we know of has as much potential to rejuvenate aging human cells.

Chinese Tradition and Modern Science. The Heart of T.A. Sciences

In 2000 scientists at leading edge California Biotech firm, Geron, were able to find the active ingredient from the root of the Astragalus plant. Astragalus has been recognized for over 1000 years by Doctors of Traditional Chinese Medicine for its ability to slow the signs of the aging process. They singled out one single, potent molecule from the Astragalus plant's complex molecular structure: TA-65. After isolating this molecule, Geron scientists were able to verify its ability to "wake up" a "sleeping" gene that has the ability to rejuvenate cells.

Although T.A. Sciences' introduction of TA-65 may come as a result of the 2000 year history of Chinese traditional practices, extracting the active ingredient into a pure and highly potent cellular D-aging product is a modern scientific breakthrough which we forecast will dramatically and powerfully alter our ability to slow cellular aging.**

The Solution: TA-65

TA-65 when introduced into a cell turns on a gene called hTERT, and that activates an enzyme called telomerase. Accordingly, this molecule is a "telomerase activator" and hence the name of our company: Telomerase Activation Sciences (T.A. Sciences). The potent telomerase activator, TA-65, is at the heart of our mission to increase your Healthspan: TA-65 helps restore the youthful, functional activity lost due to aging or stress, essentially giving cells a new lease on life. The physical decline that comes with the passage of time is primarily the result of cellular wear and tear. Young, healthy cells maintain themselves, but as we age, cells lose the capacity for rejuvenation.

T.A. Sciences' goal is to delay the cellular degradation that marks the aging process. When TA-65 activates the enzyme telomerase, it causes the ends of the chromosomes which have been shortened by age and stress to become long and youthful again. These chromosomal ends are telomeres. Telomeres are DNA sequences that protect the integrity of the genetic material located on the rest of a chromosome; they serve as genetic "buffers." The result of elongating telomeres is that cells which would normally degrade continue to thrive. Below is a more in-depth discussion of telomeres and telomerase.

Telomerase Activation extends the lifespan of cells without altering their normal function. T.A. Sciences has pioneered an exclusive protocol to slow cellular aging.

Telomeres and the Aging Process:

Please see Professor Richard Cawthorn's University of Utah website for a good background introduction to this topic.http://learn.genetics.utah.edu/features/telomeres/

What Happens When Telomeres Shorten?

Studies have shown that longer telomeres promote longevity. (FN: learn.genetics.utah.edu). Geneticist Richard Cawthon and his team at the University of Utah found that individuals over 60 years of age with longer telomeres experienced greater heart and immune system health than their age-matched counterparts with short telomeres.

A shortened Telomere has been compared to a burning fuse on a bomb. When intact and long, they prevent chromosome ends from fraying and sticking to one another which scrambles an organism's genetic information. Telomeres that are short and frayed (due to multiple divisions) are the result of stress and the aging process.

At birth our blood cells have 8,000 base pairs of Telomeres (the length of one section is made of six base pairs of TTAGGG telomeric DNA). As we grow and age many of our cells are continually dividing. On the average we lose about 50 base pairs per year, except in areas of stress, where the loss can be significantly higher. The result is that by the time we are elderly, telomere length decreases to 3 or 4,000 base pairs. In individuals who have suffered lots of stress, base pair length can get down to 1,500.

Telomeres must be a certain length in order to continue to divide and grow new skin, blood, bone and other cells as we need them. Cells with short telomeres stop dividing. The result can be the various conditions associated with old age.